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据估算，英国每年有2500人患上心碎综合征，这种综合征还可能引发与心脏病并发症类似的病症。目前尚不清楚是什么导致了心碎综合征，但据了解，丧亲之痛、车祸、地震甚至婚礼等喜事急性应激引起的肾上腺素急剧飙升，会导致部分心壁运动丧失，进而诱发急性心衰。
Two molecules – called microRNA-16 and microRNA-26a — that are linked to depression, anxiety and increased stress levels had previously been detected in the blood of takotsubopatients. Researchers assessed the impact of exposing cells from human hearts (taken fromorgans that were unsuitable for transplants) and rat hearts to the two molecules.
此前在心碎综合征患者的血液中检测到两种名为微小核糖核酸-16和微小核糖核酸-26a的分子，这两种分子与抑郁、焦虑和压力水平升高有关。研究人员评估了将人类心脏（取自不适合移植的器官）和大鼠心脏的细胞暴露于这两种分子的影响。
Afterwards, both sets of heart cells were more sensitive to adrenaline, they wrote in thejournal Cardiovascular Research.
他们在《心血管研究》杂志发表的文章中称，随后发现，这两组心脏细胞对肾上腺素更敏感。
In patients with takotsubo, the bottom of the heart stops beating, and the top of the heartbeats more, said the lead study author, Dr Liam Couch from Imperial College London. “Basically, we found the exact same thing happens when we increase the exposure to themolecules [in an experimental setting]. It reproduced exactly what happens in takotsubo, so it made it more likely for the takotsubo to occur.”
该研究的主要作者、伦敦帝国理工学院的利亚姆·考奇博士称，心碎综合征患者的心脏底部停止跳动，顶部跳动更多。“基本上，我们发现（在实验环境中）当我们增加心脏对这两种分子的暴露时，会发生完全相同的情况。它准确地再现了心碎综合征发生的情况，因此使心碎综合征发生的可能性更大。”
Overall, the findings appeared to link long-term stress and the dramatic takotsubo responseto a sudden shock.
总的来说，研究结果似乎显示了长期的压力与因意外打击引发的突发性心碎综合征之间的关联。
However, the problem is that it is not possible, for now, to diagnose takotsubo in patientsbefore it happens, making it difficult to test whether these molecules are elevated in real-lifecases, Couch explained.
然而，考奇解释称，问题是，目前还不能在心碎综合征发生之前对患者进行诊断，这使得在现实中很难检测这些分子是否升高。
